Neurologic Sequellae of Nutritional Deficiencies 01

Topic: Imaging

Created on Saturday, September 22 2007 by jdmiles

Last modified on Saturday, September 22 2007.

 
        A) Ataxia, nystagmus, jaundice, and insomnia
 
        B) Ataxia, telangiactasias, dementia, and hallucinations
 
        C) Ophthalmoparesis, nystagmus, confusion, and ataxia
 
        D) Confabulation, nystagmus, vertigo, and nausea
 
        E) Confusion, nuchal rigidity, headache, and nausea
 

 

	= Go back to the top of the page.
	= Reload a different version of this question ().
	= Load a random question from the database.
	= Use this question as a template to create a totally NEW question.
	= Enter detailed rating for this question!
	= How users like you have rated this question.

This question was created on September 22, 2007 by jdmiles.
This question was last modified on September 22, 2007.

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

ANSWERS AND EXPLANATIONS

---

A) ataxia, nystagmus, jaundice, and insomnia

This answer is incorrect.

This FLAIR MRI shows hyperintensity in the medial thalamic nuclei. This finding is characteristic of both Wernicke encephalopathy and Korsakoff dementia. The typical symptoms of Wernicke encephalopathy include ophthalmoparesis, nystagmus, ataxia, and a change in mental status characterized by confusion and apathy. Wernicke encephalopathy and Korsakoff dementia are both due to a deficiency of thiamine, and are both more common in alcoholics. Treatment of Wernicke encephalopathy requires administration of Thiamine.  Adams and Victor recommend 50mg IV and 50mg IM immediately, followed by 50mg IM daily.  (See References)

Please log in if you want to rate questions.

 

 

 

 

 

 

 

 

 

---

B) ataxia, telangiactasias, dementia, and hallucinations

This answer is incorrect.

This FLAIR MRI shows hyperintensity in the medial thalamic nuclei. This finding is characteristic of both Wernicke encephalopathy and Korsakoff dementia. The typical symptoms of Wernicke encephalopathy include ophthalmoparesis, nystagmus, ataxia, and a change in mental status characterized by confusion and apathy. Wernicke encephalopathy and Korsakoff dementia are both due to a deficiency of thiamine, and are both more common in alcoholics. Treatment of Wernicke encephalopathy requires administration of Thiamine, 50mg IV once ini addition to 50mg IM daily.  (See References)

Please log in if you want to rate questions.

 

 

 

 

 

 

 

 

 

---

C) ophthalmoparesis, nystagmus, confusion, and ataxia

This answer is correct.

This FLAIR MRI shows hyperintensity in the medial thalamic nuclei. This finding is characteristic of both Wernicke encephalopathy and Korsakoff dementia. The typical symptoms of Wernicke encephalopathy include ophthalmoparesis, nystagmus, ataxia, and a change in mental status characterized by confusion and apathy. Wernicke encephalopathy and Korsakoff dementia are both due to a deficiency of thiamine, and are both more common in alcoholics. Treatment of Wernicke encephalopathy requires administration of Thiamine.  Adams and Victor recommend 50mg IV and 50mg IM immediately, followed by 50mg IM daily.  (See References)

Please log in if you want to rate questions.

 

 

 

 

 

 

 

 

 

---

D) confabulation, nystagmus, vertigo, and nausea

This answer is incorrect.

This FLAIR MRI shows hyperintensity in the medial thalamic nuclei. This finding is characteristic of both Wernicke encephalopathy and Korsakoff dementia. The typical symptoms of Wernicke encephalopathy include ophthalmoparesis, nystagmus, ataxia, and a change in mental status characterized by confusion and apathy. Wernicke encephalopathy and Korsakoff dementia are both due to a deficiency of thiamine, and are both more common in alcoholics. Treatment of Wernicke encephalopathy requires administration of Thiamine.  Adams and Victor recommend 50mg IV and 50mg IM immediately, followed by 50mg IM daily.  (See References)

Please log in if you want to rate questions.

 

 

 

 

 

 

 

 

 

---

E) confusion, nuchal rigidity, headache, and nausea

This answer is incorrect.

This FLAIR MRI shows hyperintensity in the medial thalamic nuclei. This finding is characteristic of both Wernicke encephalopathy and Korsakoff dementia. The typical symptoms of Wernicke encephalopathy include ophthalmoparesis, nystagmus, ataxia, and a change in mental status characterized by confusion and apathy. Wernicke encephalopathy and Korsakoff dementia are both due to a deficiency of thiamine, and are both more common in alcoholics. Treatment of Wernicke encephalopathy requires administration of Thiamine, 50mg IV once ini addition to 50mg IM daily.  (See References)

Please log in if you want to rate questions.

 

 

 

 

References:

1. Victor, M., and Ropper, A.H. (2001). Adams and Victor's Principles of Neurology, 7th Edition. McGraw-Hill, New York.
2. Rowland, L.P. (Ed) (2000). Merritt's Neurology, 10th Edition. Lippincott Williams & Wilkins, Philadelphia.
3. So, Y.T., and Simon, R.P. (2004). Deficiency diseases of the nervous system. In Bradley, W.G., Daroff, R.B., Fenichel, G.M., and Jankovic, J. (Eds.). Neurology in Clinical Practice, Fourth Edition. Butterworth Heinemann, Philadelphia, pp. 1693-1708.

Please log in if you want to rate questions.

 

FrontalCortex.com -- Neurology Review Questions -- Neurology Boards -- Board Review -- Residency Inservice Training Exam -- RITE Exam Review
imaging
Neurologic Sequellae of Nutritional Deficiencies 01
Question ID: 092207197
Question written by J. Douglas Miles, (C) 2006-2009, all rights reserved.
Created: 09/22/2007
Modified: 09/22/2007
Estimated Permutations: 5400