Have you seen the parietal watch?

Neurologic Sequellae of Nutritional Deficiencies 01

Topic: Imaging

Created on Saturday, September 22 2007 by jdmiles

Last modified on Saturday, September 22 2007.

This MRI most likely comes from a patient with which of the following findings?


 
        A) Confusion, nuchal rigidity, headache, and nausea
 
        B) Ophthalmoparesis, nystagmus, ataxia, and confusion
 
        C) Seizures and ataxia
 
        D) Ataxia, nystagmus, jaundice, and insomnia
 
        E) Ataxia, telangiactasias, dementia, and hallucinations
 

 


Back to the question = Go back to the top of the page.
See another question like this one = Reload a different version of this question ().
Click here for a random question = Load a random question from the database.
Clone this question = Use this question as a template to create a totally NEW question.
Rate this question = Enter detailed rating for this question!
Average user rating for this question = 4.5 = How users like you have rated this question.
This question was created on September 22, 2007 by jdmiles.
This question was last modified on September 22, 2007.

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

ANSWERS AND EXPLANATIONS




A) confusion, nuchal rigidity, headache, and nausea

This answer is incorrect.


This FLAIR MRI shows hyperintensity in the medial thalamic nuclei. This finding is characteristic of both Wernicke encephalopathy and Korsakoff dementia. The typical symptoms of Wernicke encephalopathy include ophthalmoparesis, nystagmus, ataxia, and a change in mental status characterized by confusion and apathy. Wernicke encephalopathy and Korsakoff dementia are both due to a deficiency of thiamine, and are both more common in alcoholics. Treatment of Wernicke encephalopathy requires administration of Thiamine, 50mg IV once ini addition to 50mg IM daily.  (See References)

Back to the questionSee another question like this oneClick here for a random questionClone this question Rate this questionAverage user rating for this question = 4.5
Please log in if you want to rate questions.

 

 

 

 

 

 

 

 

 




B) ophthalmoparesis, nystagmus, ataxia, and confusion

This answer is correct.


This FLAIR MRI shows hyperintensity in the medial thalamic nuclei. This finding is characteristic of both Wernicke encephalopathy and Korsakoff dementia. The typical symptoms of Wernicke encephalopathy include ophthalmoparesis, nystagmus, ataxia, and a change in mental status characterized by confusion and apathy. Wernicke encephalopathy and Korsakoff dementia are both due to a deficiency of thiamine, and are both more common in alcoholics. Treatment of Wernicke encephalopathy requires administration of Thiamine.  Adams and Victor recommend 50mg IV and 50mg IM immediately, followed by 50mg IM daily.  (See References)

Back to the questionSee another question like this oneClick here for a random questionClone this question Rate this questionAverage user rating for this question = 4.5
Please log in if you want to rate questions.

 

 

 

 

 

 

 

 

 




C) seizures and ataxia

This answer is incorrect.


This FLAIR MRI shows hyperintensity in the medial thalamic nuclei. This finding is characteristic of both Wernicke encephalopathy and Korsakoff dementia. The typical symptoms of Wernicke encephalopathy include ophthalmoparesis, nystagmus, ataxia, and a change in mental status characterized by confusion and apathy. Wernicke encephalopathy and Korsakoff dementia are both due to a deficiency of thiamine, and are both more common in alcoholics. Treatment of Wernicke encephalopathy requires administration of Thiamine.  Adams and Victor recommend 50mg IV and 50mg IM immediately, followed by 50mg IM daily.  (See References)

Back to the questionSee another question like this oneClick here for a random questionClone this question Rate this questionAverage user rating for this question = 4.5
Please log in if you want to rate questions.

 

 

 

 

 

 

 

 

 




D) ataxia, nystagmus, jaundice, and insomnia

This answer is incorrect.


This FLAIR MRI shows hyperintensity in the medial thalamic nuclei. This finding is characteristic of both Wernicke encephalopathy and Korsakoff dementia. The typical symptoms of Wernicke encephalopathy include ophthalmoparesis, nystagmus, ataxia, and a change in mental status characterized by confusion and apathy. Wernicke encephalopathy and Korsakoff dementia are both due to a deficiency of thiamine, and are both more common in alcoholics. Treatment of Wernicke encephalopathy requires administration of Thiamine.  Adams and Victor recommend 50mg IV and 50mg IM immediately, followed by 50mg IM daily.  (See References)

Back to the questionSee another question like this oneClick here for a random questionClone this question Rate this questionAverage user rating for this question = 4.5
Please log in if you want to rate questions.

 

 

 

 

 

 

 

 

 




E) ataxia, telangiactasias, dementia, and hallucinations

This answer is incorrect.


This FLAIR MRI shows hyperintensity in the medial thalamic nuclei. This finding is characteristic of both Wernicke encephalopathy and Korsakoff dementia. The typical symptoms of Wernicke encephalopathy include ophthalmoparesis, nystagmus, ataxia, and a change in mental status characterized by confusion and apathy. Wernicke encephalopathy and Korsakoff dementia are both due to a deficiency of thiamine, and are both more common in alcoholics. Treatment of Wernicke encephalopathy requires administration of Thiamine, 50mg IV once ini addition to 50mg IM daily.  (See References)

Back to the questionSee another question like this oneClick here for a random questionClone this question Rate this questionAverage user rating for this question = 4.5
Please log in if you want to rate questions.

 

 

 

 

References:

1. Victor, M., and Ropper, A.H. (2001). Adams and Victor's Principles of Neurology, 7th Edition. McGraw-Hill, New York.
2. Rowland, L.P. (Ed) (2000). Merritt's Neurology, 10th Edition. Lippincott Williams & Wilkins, Philadelphia.
3. So, Y.T., and Simon, R.P. (2004). Deficiency diseases of the nervous system. In Bradley, W.G., Daroff, R.B., Fenichel, G.M., and Jankovic, J. (Eds.). Neurology in Clinical Practice, Fourth Edition. Butterworth Heinemann, Philadelphia, pp. 1693-1708.
Back to the questionSee another question like this oneClick here for a random questionClone this question Rate this questionAverage user rating for this question = 4.5
Please log in if you want to rate questions.

 

FrontalCortex.com -- Neurology Review Questions -- Neurology Boards -- Board Review -- Residency Inservice Training Exam -- RITE Exam Review
imaging
Neurologic Sequellae of Nutritional Deficiencies 01
Question ID: 092207197
Question written by J. Douglas Miles, (C) 2006-2009, all rights reserved.
Created: 09/22/2007
Modified: 09/22/2007
Estimated Permutations: 5400
peer review status unavailable
rating unavailable


Top 5 tags for this item:
No tags have been created yet for this resource.
Please type in an appropriate tag for this item
more tags:
new tag:

User Comments About This Question:

0 user entries
Please log in if you'd like to add a comment.